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Fig. 3 | Allergy, Asthma & Clinical Immunology

Fig. 3

From: Co-exposure to lipopolysaccharide and desert dust causes exacerbation of ovalbumin-induced allergic lung inflammation in mice via TLR4/MyD88-dependent and -independent pathways

Fig. 3

Effects of testing samples on infiltration of inflammatory cells in the airway. a–d May-giemsa stain; bar = 20 μm. a WT ovalbumin (OVA) + heated-Asian sand dust (H-ASD) + lipopolysaccharide (LPS): moderate infiltration of eosinophils into the airway submucosa. b TLR2−/− OVA + H-ASD + LPS: moderate infiltration of eosinophils. c TLR4−/− OVA + H-ASD + LPS: slight infiltration of eosinophils. d MyD88−/− OVA + H-ASD + LPS: no significant pathological changes in the airway submucosa. Arrows show eosinophils with red granules. Triangles show tissue macrophages

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